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First HIV Serodeconversion | 1st person to go from HIV+ to HIV- to Serodeconverted in 1995 treatment created by Bio-Medical Scientist Victer Muhammad of Life Sciences of Washington Inc. Interview w/ Natures-v.com announced @ the Million Man March 10-16-95
"Understanding the Historical Record in the use of Interferon Alpha to Combat the CoronaVirus" w/ Victer Muhammad & Dr. Patrice Muhammad, Natures-v.com.
Comprehending and Arresting the Complex Mental Health Pathologies in the New Millennium Human w/ Victer Muhammad
"Simple Steps in Reducing the Affects of Myocarditis and Other Heart-related Diseases via Non-Medicinal Supplements " w/ Mr. Victer Muhammad
https://www.youtube.com/live/3SlHfrp48og?si=Vtl_SMOoALJkqcqH
In vitro studies have demonstrated that pretreatment with IFN-α/β can reduce flavivirus replication in several cell populations, including Vero cells and human foreskin fibroblasts, although treatment after infection is generally less effective (1, 13, 14, 25, 34). Moreover, mouse embryonic fibroblasts that lack the IFN-α/β receptor show increased susceptibility to WNV infection (10). However, the potential role of endogenous IFN-α/β in modulating WNV infection in vivo was untested. Our experiments demonstrate that IFN-α/β is essential for controlling WNV infection. IFN-α/βR−/− mice had an MTD of 3.8 ± 0.5 days and 100% lethality following peripheral inoculation,
The purpose of this study was to investigate the prophylactic and therapeutic potential of interferon-β (IFN-β) for use against monkeypox virus. We found that treatment with human IFN-β results in a significant decrease in monkeypox virus production and spread in vitro. IFN-β substantially inhibited monkeypox virus when introduced 6-8 h post infection, revealing its potential for use as a therapeutic. IFN-β induced the expression of the antiviral protein MxA in infected cells, and constitutive expression of MxA was shown to inhibit monkeypox virus infection.
Type 1 interferons as a potential treatment against COVID-19
Interferon-β Treatment Eliminates Cardiotropic Viruses and Improves Left Ventricular Function in Patients With Myocardial Persistence of Viral Genomes and Left Ventricular Dysfunction
Type I interferon (IFN-I) mediates tissue damage in a wide range of kidney disorders, directly affecting the biology and function of several renal cell types including podocytes, mesangial, endothelial, and parietal epithelial cells.
Interferons in the Treatment of Multiple Sclerosis
Interferon beta (IFNβ) was the first disease-modifying therapy available to treat multiple sclerosis (MS), providing patients with a treatment that resulted in reduced relapse rates and delays in the onset of disability.
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